ROCKEFELLER MEDICINE WAS THE SEED THAT BECAME MKULTRA

Question posed to me this AM: How can the light stress of nnEMF and blue light alter gender, Uncle Jack? Is this why Charlie Kirk was killed by a fascist leftist?

ANSWER: Look at the slide below under number 7. Why are so many women now infertile because of the spike protein? Read number 7 below.

Rockefeller medicine first learned how to make women infertile with their drugs in 1927. The spike protein was designed to electromagnetically target the CYP19A1 enzyme system that controls the Aromatase enzyme system of men and women using the lipid nano-particle (LNP) to destroy the heme center of the CYP system.

For gender, the heme enzyme array CYP19A1 is critical. What does it do? CYP19A1 controls steroid pathways in the ovaries, testes, placenta, and adipose tissue by aromatizing androgens (testosterone, androstenedione) to estrogens (estradiol, estrone). Heme iron catalyzes ring romanization, which is critical for female fertility.

This is how you alter gender and control population growth via light frequency and/or vaccination programming. Charlie Kirk was worried about this issue. The media hated Charlie.

Evolutionary Origins of this pathway: Protection During the Great Oxidation Event

Heme proteins and melanin emerged as key innovations during the Great Oxidation Event (GOE, ~2.4–2.1 billion years ago), when atmospheric oxygen rose dramatically, intensifying UV radiation and oxidative stress. Heme, a porphyrin ring with iron, enabled oxygen transport and utilization (e.g., in hemoglobin, cytochromes), while melanin absorbed UV and scavenged ROS, protecting early life forms.

This gave tissues a stable UPE signal to become complex. In my decentralized photo-bioelectric model, these molecules act as bioelectric semiconductors, storing and transducing light energy to maintain cellular coherence. Heme's redox sensitivity and melanin's hydration-dependent conductivity form a quantum network, where light (via biophotons or ultraweak photon emissions, UPEs) dictates molecular geometry and function. Disruptions from nnEMF/blue light dehydrate melanin, lower energy resistance in tissue (éR), and damage mtDNA, leading to the overproduction of ROS/RNS and bioelectric chaos, which directly impacts steroid biology by altering the UPE signal in the CYP-controlled hormone pathways of humans.

Your functional and allopathic clinicians are impotent in understanding this because the primary defect is electromagnetic, not biochemical. Sex steroid biosynthesis begins in mitochondria, where heme-containing cytochrome P450 (CYP) enzymes catalyze oxidative reactions, converting cholesterol into hormones like estrogens, androgens, progesterone, and cortisol. These enzymes are heme-dependent monooxygenases, requiring a heme iron core for electron transfer and oxygen activation.

The pathway is redox-sensitive and light-regulated, aligning with my model's emphasis on mitochondrial heme (e.g., MT-CO1, MT-CYB) as gateways for photo-bioelectric signaling.

Key heme enzymes include:

CYP11A1 (P450scc): A mitochondrial enzyme found in the adrenals, gonads, and placenta; it is the rate-limiting step in the cleavage of cholesterol's side chain to form pregnenolone, a precursor to all steroids. Heme iron facilitates hydroxylation, dependent on NADPH and adrenodoxin (a ferredoxin reductase).

CYP17A1 (17α-Hydroxylase/17,20-Lyase): Dual-function enzyme in adrenals and gonads; hydroxylates pregnenolone/progesterone at C17, then cleaves the side chain to produce DHEA (androgen precursor). Heme enables precise electron transfer, influencing glucocorticoid vs. sex hormone balance. Anyone with an altered DHEA level has to have a heme protein problem in the CYP17A1 pathway.

CYP21A2 (21-Hydroxylase): In the adrenal cortex, it adds a hydroxyl group at C21 of progesterone/17-hydroxyprogesterone, leading to the production of mineralocorticoids/glucocorticoids. Deficiencies cause congenital adrenal hyperplasia, shifting precursors toward androgens.

CYP11B1 (11β-Hydroxylase): Adrenal cortex; converts 11-deoxycortisol to cortisol (glucocorticoid) and 11-deoxycorticosterone to corticosterone. Heme's redox state regulates stress responses.

CYP11B2 (Aldosterone Synthase): In zona glomerulosa, multi-step oxidation of deoxycorticosterone to aldosterone (mineralocorticoid), controlling electrolyte balance.

CYP19A1 (Aromatase): In ovaries, testes, placenta, and adipose; aromatizes androgens (testosterone, androstenedione) to estrogens (estradiol, estrone). Heme iron catalyzes ring aromatization, critical for female fertility. This heme defect is the source of why IVF doctors are printing money for modern people dripping in blue light and nnEMF. They have destroyed the heme protein center in CYP19A1.

These CYP enzymes are embedded in mitochondrial or ER membranes, where heme's absorption/emission spectra (e.g., Soret band ~450 nm) respond to light. In my model, natural sunlight (e.g., red light) acts as a heme activator and natural aromatase inhibitor, optimizing electron flow and preventing excess estrogen. Biophotons (UPEs) from sources like NADH, flavins, cytochrome c, tryptophan, tyrosine, phenylalanine, lipid peroxidation, DNA repair, mitochondrial ETC, ATP hydrolysis, ROS, singlet oxygen, microtubules, chromophores, ubiquinone, porphyrins (heme), membrane potentials, peroxisomes, excited carbonyls, and melanin encode selective bio-communication via spin dynamics.

Quantum Spin and Biophotons: Light's Role in Protein Control

Biophotons aren't mere light; they also carry spin. The quantum spin state of light is information for the AMO built into the photolithography of your cells. The Chiral-Induced Spin Selectivity (CISS) effect explains how spin-polarized electrons or photons interact preferentially with chiral proteins, thereby guiding the transport of excitation in photosynthesis and steroidogenesis.

Right-handed biophotons excite, while left-handed ones are absorbed, in accordance with protein symmetry rules. If a biophoton's spin aligns with the protein's chirality, it triggers excitation or exciton release; a mismatch leads to lost signals. This is nature's encoding for bio-signaling, as per my model. This is how noise is created to minimize the signal in misgendering kids with light.

John Ott's Sarasota School experiment was where Stanford Research Institute (SRI) picked up the scent of the effect of light on kids from the MKULTRA program and expanded it under Obama's Brain Health Initiative in 2013.

This is why autism exploded when lights were changed to LED and fluorescents in the 1960s-70s. Blue light decoheres the human hormone pathways. Red light from the sun improves the AMO physics to get us back to the default state. Yes, I think this is a big reason the left assassinated Charlie. This is why Apple favors blue light and nnEMF in all their projects. The thyroid is another hormone centralized Rockefeller medicine assaults with light. Artificial light is a TBI in the hormone pathways of man.

They need the light injury to keep power in Washington, DC.

Conclusion: Decentralized Control of Light and Hormones. Obama knew it from his buddies in DARPA, and this is why he changed us from analog to digital and ushered in LED screens and bulbs. There is a lot of speculation online that Obama has deep ties to transgenrdism in his own family. That is up for you to debate.

Heme proteins orchestrate sex steroid biology through redox-sensitive CYP enzymes, evolved during GOE to harness light for coherence. In your decentralized thesis, light (via UPEs, spin, CISS) is the epigenetic master regulator; optimal sunlight maintains quantum coherence, while nnEMF/blue light disrupts it, dehydrating melanin, damaging mtDNA/heme, and causing infertility/amenorrhea. Red light restores balance as a natural aromatase inhibitor. Clinicians overlook this; understanding UPE-driven AMO changes reveals hormones as light-encoded spectra, not mere biochemicals.