1. Presentation on
Acute Abdomen
& its Management
Presented by-
• Naima Jahan
• Marjana Jannat
• Zannatul Mysha
• Ashief Ahmed
• Tafjul Islam
• Sadiazzaman juthi
• Samira Chowdhury
• Joy Prokash
• Tofayel Ahmed
• Mustafizur Rahman
2. What is acute abdomen?
Acute abdomen a clinical scenario in which a patient presents with
an intra-abdominal pathology manifesting with abdominal pain or
obstruction,which warrant urgent investigation and treatment,often
including parental fluid and sometimes surgery.
It is the most common presenting Surgical Emergency
Prepared by marj
5. Pathophysiology of acute abdomen
1) Inflammation: localized or generalized inflammation causes pain due to irritation of
the peritoneum
Eg- appendicitis, cholecystitis, diverticulitis, PID,pancreatitis,acute pyelonephritis
2) Obstruction: central colicky abdominal pain is a classical presentation of small bowel
obstruction.
Eg- intestinal obstruction, volvulus,gallstone ileus,intussusception,ureteric stone,
3)Rupture & perforation of organ:Trauma,ischemia or tissue ulceration may cause
perforation with resulting in severe abdominal pain.
Eg- perforated peptic ulcer ,perforated diverticulum , rapture aortic aneurysm,
4)Trauma-blunt & penetrating abdominal trauma etc.
6. Clinical symptoms
1 . Abdominal Pain
2. Associated Gastrointestinal Symptoms-
Nausea, vomiting, Diarrhoea,constipation, abdominal distention .
3. Systemic symptoms-
Fever, tachycardia ,hypotension, shock,Malaise, chills.
■ The primary symptom of the "acute abdomen" is –Abdominal pain
7. Types of Abdominal Pain:
1. Visceral
Due to stretching of fibers innervating the walls of hollow or solid organs.
2. Parietal
Caused by irritation of parietal peritoneum.
3. Referred pain
Pain is felt at a site away from the pathological organ
12. Management of acute abdomen
• Initial assessment
• History
• Thorough physical examination
Diagnosis can be made most of the time by a good history and a
proper physical examination.
An exact diagnosis often impossible to make after the initial assessment, and
often relying on further investigation
13. 1. Initial assessment
Airway: Ensure it's clear.
Breathing: Check respiratory rate, oxygen saturation. Give O2 if needed.
Circulation: Monitor pulse, BP. Start IV line, give fluids (usually normal saline or Ringer's
lactate).
14. 2. History & clinical examination
History of pain:
● Onset:
Sudden: perforation of bowel.
Rapid- inflammation like Appendicitis, cholecystitis.
Slow insidious onset: obstruction,malignancy
● Duration:
Less than 24 hours
● Characteristics:
Aching-dull pain poorly localized
Burning- peptic ulcer symptoms
Stabbing-ureteric colic
Gripping - smooth muscle spasm e.g. intestinal obstruction worse by movement
16. ● Aggravating & relieving factors
Movement/motion worsens parietal pain (peritonitis); lying still
brings relief
Colicky pain: Eased by vomiting or movement
Pain relief patterns:
Duodenal ulcer relieved by food
Gastric ulcer worsened by food
Biliary colic triggered by fatty meals
17. In females, abdominal pain = GYN
problem until proven otherwise
● Females
○ Last menstrual period?
○ Abnormal bleeding?
Associated symptoms
•Nausea,vomiting,fever, bowel/bladder habits, bloody discharge, change in urinary
habits, urine appearance,change in bowel habits, hematemesis, melena,regular
food/water intake.
Prepared by
18. Past history
-previous surgery, trauma,any medical diseases.
Drug history –
corticosteroid: masking pain ,anti-coagulant: intra-mural hematoma,
NSAIDS: gastritis, peptic ulcer.
Family history -colon cancer,IBD.
19. General examination
Local examination
● Inspection:
look for abdominal distension, Position of the umbilicus, Flanks full or not,
visible peristalsis, Pulsation, engorged vein,pigmentation.
● Palpation:
Check for temperature, tenderness, guarding, rigidity, rebound tenderness,
masses, organomegaly.
● Percussion:
Assess tympany, shifting dullness (ascites), and percussion tenderness.
● Auscultation:
Note bowel sounds - absent, decreased, or increased.
3.Examination:
20. 3. Investigations
Blood:
CBC, electrolytes, liver/renal function, amylase/lipase,serum creatinine,
serum -hCG (in females).
ẞ
Imaging:
•X-ray abdomen (erect): For air under diaphragm (perforation), obstruction signs.
•Ultrasound (USG): Gallstones, ectopic pregnancy.
•CT scan: If diagnosis unclear-more sensitive for appendicitis, pancreatitis, abscess.
•MRI
Urine Test:
Routine exam, pregnancy test.
21. 4.Supportive management
● NPO (nothing per oral) until further instructions.
● IV Fluids: Maintain hydration.
● NG Tube: If vomiting/severe distension.
● Urinary Catheter: For monitoring urine output if needed.
● Analgesic: Usually opioids (e.g. tramadol, morphine).
● Antibiotic: Broad-spectrum IV antibiotics if infection suspected (e.g.
ceftriaxone,penicillin etc).
● Antiulcerant
● Antiemetic
22. 5.Monitoring & follow up
Monitor vitals, urine output, and lab values regularly.
Reassess diagnosis if condition deteriorates.
23. Surgical treatment
• Appendicitis: appendisectomy
• Cholecystitis: open or laparoscopic cholecystectomy
• Peptic ulcer disease/Duodenal ulcer: vagotomy;
Billroth -2 gastrectomy; gastrojejunostomy
• Pyloric stenosis: pyloromyotomy
• Intestinal obstruction: bowelresection &end to end anastomosis
•Trauma. : damage control surgery
•Perforated peptic ulcer: closure& omental patch(graham’s patch
Prepared by marjana
25. Symptoms Differential diagnosis
Fever,pain in epigastrium,
radiate to back, relief by
leaning forward.
Acute pancreatitis
Fever, Burning intermittent pain
in epigastrium temporary relief
by food and antacid
Peptic ulcer disease
Fever, vomiting, reffered pain
in tip of the right shoulder
Acute cholecystitis
28. Appendicitis :
Inflammation of Appendix is called appendicitis.
Acute appendicitis is the most common cause of acute
abdomen in young adults
29. Etiology:
There is no unifying hypothesis regarding the etiology of
acute appendicitis.Decreased dietary fiber and increased
consumption of refined carbohydrate may be important. The
incidence of appendicitis is lowest in societies with a high
dietary fiber intake. In resource- poor countries that are
adopting a more refined western type diet, the incidence
continues to rise.
30. Causes:
● Foreign body
● Fecolith (hard stool)
● Trauma
● Immunocompromised (infection)
● Unknown/Idiopathic
Symptoms:
●Periumbilical colic
● Pain shifting to the right iliac fossa
● Anorexia
● Nausea
● Fever
31. Clinical signs in appendicitis :
● Pyrexia
● Localised tenderness in the right iliac fossa
● Muscle guarding
● Rebound tenderness
Signs to elicit in appendicitis:
● Pointing sign
● Rovsing’s sign
● Psoas sign
● Obturator sign
34. Complications:
1) Appendicular lump
2) Appendicular abcess
3) Perforation
4) Peritonitis
5) Septicemia
6) Gangrenous Apenndicitis
Investigation
The diagnosis of acute appendicitis is essentially clinical.
37. Treatment :
Conservative:
● NPO until further instructions
● IV fluids
● IV antibiotics
● Analgesic
● Oxygen supplemention
● Monitoring of vital sign
Surgical:
The main surgical treatment for appendicitis is appendectomy performed by
either:
● Open surgery
● Laparoscopic surgery
39. Defination:
Cholecystitis refers to the inflammation of the gallbladder.
This condition may develop suddenly, known as acute cholecystitis, or persist
over a longer period, termed chronic cholecystitis.
Commonly, it occurs in a patient with pre-existing chronic cholecystitis but
often also can occur as a first presentation.
Usual cause is impacted gallstone in the Hartmann's pouch, obstructing cystic
duct.
Cholecystitis
40. Types
Acute chelecystitis is two types:
● Calculus Cholecystitis
Refers to gallbladder inflammation that is directly associated with the presence
of gallstone.
● Acalculus Cholecystitis
Refers to inflammation of the gallbladder occurring without the presence
gallstone or any blockage in the cystic duct.
41. Clinical features:
● Pain in the right hypochondrium, initially colicky than constant.
● Pain reffered to back, right scapula or tip of right shoulder.
● Palpable, tender, smooth, soft gallbladder.
● Fever, nausea, palpable tender mass in GB region (25%).
● Muscle guard & muscle rigidity in abdomen.
● Area of hyperaesthesia between 9th and 11th ribs posteriorly on the right
side (Boas's sign).
● Murphy's sign may be positive.
43. Investigation:
● Blood for TC,DC- Neutrophilic leucocytosis.
●Ultrasound abdomen-Very useful, reveals presence or absence of gallstones
and thickening of gallbladder wall. Sonographic Murphy's sign may be positive.
●Plain X-ray abdomen-10% of gallstones are radio-opaque; also rules out other
causes of acute pain abdomen. Gas is seen in emphysematous GB.
●CT scan is useful in identifying the perforation, impacted stone, gallbladder
wall thickness and oedema.
48. Definition:
Acute inflammation of the pancreas is called acute pancreatitis.
Pathogenesis of acute pancreatitis :
Pancreatic duct obstruction edema Compression of blood vessel Ischemia
⇨ ⇨ ⇨ ⇨
acinar cell injury release of pancreatic & lysosomal enzymes activation of
⇨ ⇨
enzymes autodigestion of pancreas.
⇨
Pancreatitis.
49. Etiology:
1.Gall stone(most common)
2.Alcohol
3.Trauma(Blunt,Iatrogenic ERCP)
⇨
4.Biliary tract disease
5.Drugs-Halothane,Steroids,antipsychotic drugs.
Management of acute pancreatitis :
1) Clinical Features-
i. Sudden severe epigastric pain radiating to the back.
ii. Pain exaggerated after fatty meal & Relived by leaning forward position.
iii. Nausea,Vomiting,Fever.
50. 2)Investigation -
i. Biochemical test :
Serum amylase-↑, *Serum lipase-↑
ii. Imaging :
a. Ultrasonogram of whole abdomen special attention to hepatobiliary system with
pancreas
b. CT scan
iii. Hematological :
Complete Blood Count(CBC)
## To predict the severity of acute pancreatitis there is 2 types of scoring-
Ranson & Glasgow scoring.
51. 3) Treatment-
Conservative
i. NPO(nothing per oral) until further instructions
ii. IV fluid
iii. Oxygen supplementation
iv. Analgesic to relief pain
v. Antibiotic to prevent secondary infections
vi. Monitoring of the vital signs
vii. Catheterization to measure intake & output
**HDU transfer may be needed.
52. #Two clinical signs seen in
severe(hemorrhagic) pancreatitis:
1)Cullen’s sign- discoloration around
the central umbilicus.
2)Grey Turner's sign- discoloration
in & around the flanks.
55. Causes of Intestinal Obstruction
A. Dynamic
Intra luminal-
Fecal infection,
Foreign body,
Gall stone,
Bezoar
Intra mural-
Volvulus,
Stricture,
Malignancy
Extra mural-
Band,
Adhesion,
Obstruction
B. Adynamic-
Paralytic ileus,
Pseudoobstruction,
Mesenteric vascular occlusion
57. Investigations:
1) Plain x-ray of abdomen in erect posture with both dome of diaphragm.
2) Ultrasonogram of whole abdomen
3) CT-scan
4) CBC
5) Serum electrolyte
6) Serum creatinine
58. Fig: X-ray of small bowel obstruction
Prepared by SAMIRA
In small bowel
obstruction, Multiple
gas fluid level in the
centre of abdomen &
Valvulae conniventes
(multiple ring like
radio opaque shadow)
seen
59. Fig: X-ray of Large bowel obstruction
Prepared by SAMIRA
In large bowel
obstruction,Mul
tiple gas fluid at
the periphery &
haustral fold are
seen.
60. Treatment:
1.Nothing by mouth until further instructions
2.Gastrointestinal drainage via nasogastric tube
3.Fluid & electrolyte replacement
4.Antibiotics
5.Catheter
6.Relief of obstruction:
Usually surgical, surgical treatment is necessary for most cases of intestinal
obstruction but should be delayed until resuscitation is complete, provided there is
no sign of strangulation or evidence of closed-loop obstruction
61. • Steady, well-localized epigastric or
LUQ pain
• Described as a “burning”,
“gnawing”, “aching”
• Increased by coffee, stress, spicy
food, smoking
• Decreased by alkaline food, antacids
Prepared by
Pain due to -
Duodenal ulcer relieved by food
Gastric ulcer worsened by food
Peptic Ulcer Disease
62. Fig- xray of a perforated peptic ulcer
Cresentic gas shadow under
right dome of diaphragm
Prepared by marjana
64. D) Surgical treatment:
For gastric ulcer-
1.Bilroth-1 gastrectomy removal of distal
⇨
portion of the stomach
2.Bilroth-2 gastrectomy removal of lower
⇨
portion of the stomach
For duodenal ulcer-
1.Truncal vagotomy
2.selective vagotomy
3.Highly selective vagotomy
Prepared by SAMIRA
65. Ureteric colic
● Severe, sudden flank pain due to
urinary tract obstruction, usually by a
kidney stone.
● Felt in the flank (costovertebral angle/
renal angle) and radiates to groin,
scrotum in males, or labia in
females,thigh.
Investigations:
• Urinalysis: hematuria.
•Non-contrast CT KUB (investigation of choice).
•Ultrasound for hydronephrosis.
•Plain xray of KUB region
Prepared by marja
66. Management
1. Initial Management
• Pain relief (first line): NSAIDs (e.g. diclofenac IM or IV).
• If pain persists: Add opioids (e.g. morphine) if NSAIDs are insufficient.
2. Hydration
3. Medical Expulsive Therapy (for stones <10 mm)
Drink plenty of water,joulting movement
Alpha blockers: e.g. tamsulosin to facilitate stone passage.
4.definate Management ( if stone doesn't pass or complications arises)
ESWL (Extracorporeal Shock Wave Lithotripsy): For stones <2 cm.
URS (Ureteroscopy) + Laser lithotripsy: For mid/distal ureteric stones.
PCL (Percutaneous urolithotomy): For large (>2 cm) renal
stones.
5. Emergency Intervention - ureterolithotomy
68. Ectopic pregnancy
● Presents with sudden lower
abdominal pain, amenorrhea,
and vaginal bleeding.
● If ruptured, it can lead to
hemoperitoneum and shock,
mimicking other surgical
emergencies.
● A common cause of acute
abdomen in reproductive-age
women.
● Investigation:
i) Blood:Hb%,TC,DC,ESR
ii) Blood grouping & Rh typing
iii) Transvaginal sonography
Treatment: Surgical
(laparoscopic removal) or
medical (methotrexate) if
unruptured.
● Diagnosis:
○ Positive B-hCG,
○ transvaginal USG
shows empty uterus.
69. ##Case Presentation:
A 60-year-old woman presents to the emergency department with sudden onset of
severe epigastric pain radiating to the back, which started 8 hours ago. The pain is
constant and worsens when lying flat. She has had multiple episodes of vomiting and
appears distressed. She reports no history of alcohol consumption, trauma, or recent
medication changes.
*On examination:
•Pulse: 108 bpm
•BP: 98/60 mmHg
•Temperature: 101°F (38.3°C)
•Abdomen: Epigastric tenderness, reduced bowel sounds
•No hepatosplenomegaly, no guarding
•Mild icterus present
70. ☞ Questions:
1)What is the most likely diagnosis?
2)What is the most probable underlying cause in this patient?
3)What are the signs & symptoms suggesting your diagnosis in this
patient?
4)Mention two clinical signs that suggest severity of this disease.
5)Which investigations would you order to confirm the diagnosis and
identify the cause?
6)How would you manage this patient initially?
7)What complications would you monitor for in the next few days?
71. Case presentation
Suppose you are an intern/Mo in a busy hospital. You are referred to a 29 year old
woman with a severe and unremitting periumbilical pain.The pain has been worsening
for the past 2 days and now localised to the right illliac fossa.
On examination:
BP 105/64 mmHg;
temperature 38.3° C;
RR 18/mins and
Saturation: 97%
72. ☞ Questions:
1)What is the most likely diagnosis?
2)What are the differential diagnosis in this patient?
3)What are the signs & symptoms suggesting your diagnosis in this
patient?
4)Which investigations would you order to confirm the diagnosis and
identify the cause?
5)How would you manage this patient initially?
6)What are the complications?