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Non Surgical intervention in ASD
           and VSD
ASD
ASD
Atrial Septal Defect (ASD)
ASD
•   An atrialseptal defect (ASD) is a hole in the wall between the two
    upper chambers of your heart.

•   The condition is present from birth (congenital).

•   Smaller atrialseptal defects may close on their own during infancy or
    early childhood.

•   Large and long-standing atrialseptal defects can damage your heart
    and lungs. An adult who has had an undetected atrialseptal defect
    for decades may have a shortened life span from heart failure or
    high blood pressure in the lungs. Intervention is usually necessary
    to repair atrialseptal defects to prevent complications.
Causes of Atrial Septal Defect (ASD)
•   Doctors know that heart defects present at birth (congenital) arise
    from errors early in the heart development, but there is often no
    clear cause. Genetics and environmental factors may play a role.

•   An atrialseptal defect allows freshly oxygenated blood to flow from
    the left upper chamber of the heart (left atrium) into the right upper
    chamber of the heart (right atrium). There it mixes with
    deoxygenated blood and is pumped to the lungs, even though it is
    already refreshed with oxygen.

•   If the atrialseptal defect is large, this extra blood volume can overfill
    the lungs and overwork the heart. If not treated, the right side of the
    heart eventually enlarges and weakens. In some cases, the blood
    pressure in your lungs increases as well, leading to pulmonary
    hypertension.
Symptoms of Atrial Septal Defect
                (ASD)
•   Many babies born with atrialseptal defects do not have
    signs or symptoms. In adults, signs or symptoms may
    not develop until age 30 or later.

•   Your doctor may first uncover an atrialseptal defect
    during a regular checkup while listening to your heart
    using a stethoscope. Hearing a heart murmur may signal
    a hole in your heart.
•   Atrialseptal defects are often found when an ultrasound
    exam of the heart (echocardiogram) is done for another
    reason.

•   Signs and symptoms of atrialseptal defects develop once
    damage occurs to the heart and lungs. Infants with larger
    atrialseptal defects may have poor appetites and not
    grow as they should. Adults and infants may have signs
    of heart failure or arrhythmias.
When to seek medical advice for
    Atrial Septal Defect (ASD) :


Poor appetite                     Failure to gain
                                      weight
                              2




                  1
                                       Bluish
Shortness of                      discoloration of
                  2
                          2




  breath                              the skin



   Easy tiring
   Swelling of        2




                                  skipped beats
                          2




  legs, feet or
    abdomen
Morphology
                    MORPHOLOGY

Types of ASD:

Ostium primum

Ostium secundum

Sinus venosus

Coronary sinus defects



↑Left to right shunt:

* left ventricular compliance↓

* left atrial pressure ↑
Asd and vsd
Percutaneous ASD closure
          • Percutaneous closure of an ASD is
            indicated for the closure of
            secundum ASDs with a sufficient rim
            of tissue around the septal defect so
            that the closure device does not
            impinge upon the SVC, IVC, or the
            tricuspid or mitral valvs.

          • The Amplatzer Septal Occluder
            (ASO) is commonly used to close
            ASDs. The ASO consists of two self-
            expandable round discs connected
            to each other with a 4 mm waist,
            made up of 0.004–0.005´´ Nitinol
            wire mesh filled with Dacron fabric.

          • Percutaneous closure is the method
            of choice in most centres.
VSD
VSD
Morphology
                    MORPHOLOGY


Four components of Septum:

Membranous, inlet, trabecular, outlet (conal,infundibular) part.

Types of VSD :

Muscular VSD

Membranous VSD

Doubly committed subarterial VSD
Asd and vsd
Asd and vsd
Pathophysiology
                  PATHPHYSIOLOGY



                 Shunt      Qp/Qs     P/A systolic
                                      pressure ratio
Restrictive      Small      1~1.4     <0.3
VSD
Moderately      Moderat     1.4~2.2   0.3~0.66
restrictive VSD e

Nonrestrictive   Large      >2.2      >0.66
VSD
Eisenmenger      Right to   <1        1
VSD              left
Asd and vsd
Coarc Stent
  Repair
THE FUTURE




                                            IMPROVED
                                            CONTRAST
                                            AGENTS
                          Interventional
Real time 3-D imaging
                          cath



                    Improved Accuracy for
                   Repair of Complex CHD

                        Repair of Complex
                              CHD
We Care Core Values


•   “We have a very simple business model that keeps you as
    the centre.”
    Having the industry’s most elaborate and exclusive Patient
    Care and Clinical Coordination teams stationed at each of
    our hospitals, we provide you the smoothest and seamless
    care ever imagined.
Thank You
                CONTACT US:
            High Beam Global
            209, Udyog Vihar, Phase – 1, Gurgaon.
            Mobile : +91-124-4879699
            Phone: +91- 9990066728
            E-mail: nandita.gupta@highbeamglobal.com
            Web address: www.highbeamglobal.com

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Asd and vsd

  • 1. Non Surgical intervention in ASD and VSD
  • 4. ASD • An atrialseptal defect (ASD) is a hole in the wall between the two upper chambers of your heart. • The condition is present from birth (congenital). • Smaller atrialseptal defects may close on their own during infancy or early childhood. • Large and long-standing atrialseptal defects can damage your heart and lungs. An adult who has had an undetected atrialseptal defect for decades may have a shortened life span from heart failure or high blood pressure in the lungs. Intervention is usually necessary to repair atrialseptal defects to prevent complications.
  • 5. Causes of Atrial Septal Defect (ASD) • Doctors know that heart defects present at birth (congenital) arise from errors early in the heart development, but there is often no clear cause. Genetics and environmental factors may play a role. • An atrialseptal defect allows freshly oxygenated blood to flow from the left upper chamber of the heart (left atrium) into the right upper chamber of the heart (right atrium). There it mixes with deoxygenated blood and is pumped to the lungs, even though it is already refreshed with oxygen. • If the atrialseptal defect is large, this extra blood volume can overfill the lungs and overwork the heart. If not treated, the right side of the heart eventually enlarges and weakens. In some cases, the blood pressure in your lungs increases as well, leading to pulmonary hypertension.
  • 6. Symptoms of Atrial Septal Defect (ASD) • Many babies born with atrialseptal defects do not have signs or symptoms. In adults, signs or symptoms may not develop until age 30 or later. • Your doctor may first uncover an atrialseptal defect during a regular checkup while listening to your heart using a stethoscope. Hearing a heart murmur may signal a hole in your heart. • Atrialseptal defects are often found when an ultrasound exam of the heart (echocardiogram) is done for another reason. • Signs and symptoms of atrialseptal defects develop once damage occurs to the heart and lungs. Infants with larger atrialseptal defects may have poor appetites and not grow as they should. Adults and infants may have signs of heart failure or arrhythmias.
  • 7. When to seek medical advice for Atrial Septal Defect (ASD) : Poor appetite Failure to gain weight 2 1 Bluish Shortness of discoloration of 2 2 breath the skin Easy tiring Swelling of 2 skipped beats 2 legs, feet or abdomen
  • 8. Morphology MORPHOLOGY Types of ASD: Ostium primum Ostium secundum Sinus venosus Coronary sinus defects ↑Left to right shunt: * left ventricular compliance↓ * left atrial pressure ↑
  • 10. Percutaneous ASD closure • Percutaneous closure of an ASD is indicated for the closure of secundum ASDs with a sufficient rim of tissue around the septal defect so that the closure device does not impinge upon the SVC, IVC, or the tricuspid or mitral valvs. • The Amplatzer Septal Occluder (ASO) is commonly used to close ASDs. The ASO consists of two self- expandable round discs connected to each other with a 4 mm waist, made up of 0.004–0.005´´ Nitinol wire mesh filled with Dacron fabric. • Percutaneous closure is the method of choice in most centres.
  • 12. Morphology MORPHOLOGY Four components of Septum: Membranous, inlet, trabecular, outlet (conal,infundibular) part. Types of VSD : Muscular VSD Membranous VSD Doubly committed subarterial VSD
  • 15. Pathophysiology PATHPHYSIOLOGY Shunt Qp/Qs P/A systolic pressure ratio Restrictive Small 1~1.4 <0.3 VSD Moderately Moderat 1.4~2.2 0.3~0.66 restrictive VSD e Nonrestrictive Large >2.2 >0.66 VSD Eisenmenger Right to <1 1 VSD left
  • 17. Coarc Stent Repair
  • 18. THE FUTURE IMPROVED CONTRAST AGENTS Interventional Real time 3-D imaging cath Improved Accuracy for Repair of Complex CHD Repair of Complex CHD
  • 19. We Care Core Values • “We have a very simple business model that keeps you as the centre.” Having the industry’s most elaborate and exclusive Patient Care and Clinical Coordination teams stationed at each of our hospitals, we provide you the smoothest and seamless care ever imagined.
  • 20. Thank You CONTACT US: High Beam Global 209, Udyog Vihar, Phase – 1, Gurgaon. Mobile : +91-124-4879699 Phone: +91- 9990066728 E-mail: nandita.gupta@highbeamglobal.com Web address: www.highbeamglobal.com