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BURNS
Presented By:
Mr. Mwandalesa
PREPARED DR. BANDA
WISDOM CHILUFYA
MBCHB (CBU)
DEFINITION
Refers to coagulative necrosis of the
tissues caused by heat transferred from
the source to the body.
NB: frostbite is coagulative necrosis
caused by extreme degrees of cold
ETIOLOGICAL CLASSIFICATION OF BURNS
• Thermal burns: Flame and Scald burns
• Electrical Burns
• Chemical Burns
• Radiation Burns
• Inhalation Burns
• Friction Burns
• FRICET
ANATOMICAL CLASSIFICATION
1. First degree burns – erythema – redness –
sunburn – painful – n0 blister formation
[vesicle/bulla] – epidermis only
2. Second degree burns – blister formation – very
painful – damage to basement membrane –
epidermis & superficial dermis
3. Third degree burns – dermis – painless – aka =
full thickness burns
4. Fourth degree burns – subcutaneous tissue –
muscle & bone
A mixture is of the above is a common finding
DEPENDING ON THICKNESS OF SKIN INVOLVED
I. Partial thickness burns: It is either first
or second degree burn which is red and
painful, often with Blisters
II. Full thickness burns: It is third degree
burns which is charred, insensitive, deep
involving all layers of the skin.
JACKSON’S ZONES OF BURN
PATHOPHYSIOLOGY
Heat causes coagulation necrosis of skin and subcutaneous
tissue.
Release of vasoactive peptides
Altered capillary permeability
Loss of fluid
Severe hypovolemia
Decreased cardiac output
1. Decreased renal blood -Oliguria (Renal failure)
2. Altered pulmonary resistance causing pulmonary oedema
 Infection
 Systemic Inflammatory Response Syndrome (SIRS)
 Multi Organ Dysfunction Syndrome (MODS).
PHASE 1 = RAPID RESPONSE SYSTEM = NERVOUS SYSTEM
• “Fight or flee”
• Governed by hypothalamus = autonomic
nervous system [sympathetic component]
• Release of adrenaline and noradrenaline
• Pupils dilate
• Heart rate rises
• Brain alert
• Airways dilate
PHASE 2 = LOCAL RESPONSE
• Initial response is vasoconstriction by the
catecholamine augmented by endothelin =
most potent vasoconstrictor known
• Soon this is replaced by vasodilatation and
increased vascular permeability at injury site
• Principal vasodilators include nitric oxide,
prostacyclin, histamine, serotonin
• This allows for the extravasation of inflammatory
cells to come in to kill the invaders and clean up
the mess
PHASE 3 = ENDOCRINE RESPONSE
• Remember the bigger the insult or injury the bigger the
response
• Therefore limited injury will have no systemic response
• The goal of all these responses is preserve intravascular
volume
• Any insult threatening intravascular integrity
HYPOTHALAMUS-PITUITARY-ADRENAL AXIS
• Low Bp is picked up by the baroreceptors =>
hypothalamus=>corticotrophin releasing hormone=>
adrenacorticotropin hormone=> cortisol & aldosterone=>
raises sensitivity of catecholamine & sodium reabsorption
RENIN-ANGIOTENSIN-ALDOSTERONE AXIS
• Baroreceptors in the juxtaglomerular
apparatus=> renin=> angiotensinogen=>
angiotensin-1=> angiotensin converting
enzyme=>angiotensin-2=> vasoconstrictor
& stimulate aldosterone release by the
adrenal cortex
CRITERIA FOR ADMISSION TO A BURNS
CENTER
1. Burns >20% bsa in adults
2. Burns >10% bsa in children & elderly >50
3. Electrical burns
4. Chemical burns [extensive]
5. Suspected child abuse burns
6. Inhalation burns
7. Special areas – face, hands, genitalia , major joints
8. Concomitant trauma – trauma center first
9. Co-morbidities – cardiopulmonary, dm, epilepsy
10. >5% deep burns
ASSESMENT OF BURNS
1. WALLACE RULE OF 9
RULE OF PALM
• “RULE OF THE PALM” = PATIENT’S PALM IS APPROXIMATELY 1%
OF THE BSA IN BOTH ADULTS AND CHILDREN
INVESTIGATIONS
• FBC.
• U+E.
• If inhalation suspected: chest X-ray,
arterial blood gases, CO
• estimation.
• Blood group and crossmatch.
• ECG/cardiac enzymes with electrical
burns.
IN SUMMARY
1. ABCDE
2. Pain control
3. Tetanus prophylaxis
4. Secondary survey + % bsa + depth
5. Foley catheter
6. Fluid resuscitation => oral or intravenous
7. Proton pump inhibitors
8. Topical antibiotics
9. Daily wound care
10. Keep the room warm
OTHER ESGENERAL MEASUR
• High protein diet
• Blood transfusion
• Physiotherapy
• The use of a cradle
• Always keep the patient warm
FLUID RESUSCITATION
1. PARKLAND FORMULA:-
1. 4ml x KG x % BURN AREA = MLS
2. Half is administered in first 8 hours
3. The next half is administered in the following 16 hours
• This formula or any formula is not cast in stone and should be
modified to achieve the desired urine output
• Literature says don’t go beyond 8.4 liters. Maximum percentage for
fluid should be less than 50%
• But in practice (30-40%)
E.g. 22years old student at Zera sustains 20% burns, he weight is 70
calculate his fluid requirement using parkland formula
2. MUIR AND BARCLAY
• =(%BSA x Kg) /2
Fluids are given over 36 hrs. as following
3. MODIFIED BROOKES
It calculates the deficity:
=2 X %bsa x kg.
COMPLICATIONS OF BURNS
Early complications
• Airway obstruction
• Shock
• Hypoxia
• RDS
• Hypothermia
Intermediately complications
• Electrolyte imbalance
• Infection
• Anemia
• Stress ulcers (Curling ulcers)
• Paralytic ileus
• Renal failure
• Compartment syndrome
LATE COMPLICATIONS
• Contractures
• Hypertrophic scar
• keloids
• Margolis's ulcer

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BURNS.pptx lecture presentation surgery.

  • 1. BURNS Presented By: Mr. Mwandalesa PREPARED DR. BANDA WISDOM CHILUFYA MBCHB (CBU)
  • 2. DEFINITION Refers to coagulative necrosis of the tissues caused by heat transferred from the source to the body. NB: frostbite is coagulative necrosis caused by extreme degrees of cold
  • 3. ETIOLOGICAL CLASSIFICATION OF BURNS • Thermal burns: Flame and Scald burns • Electrical Burns • Chemical Burns • Radiation Burns • Inhalation Burns • Friction Burns • FRICET
  • 4. ANATOMICAL CLASSIFICATION 1. First degree burns – erythema – redness – sunburn – painful – n0 blister formation [vesicle/bulla] – epidermis only 2. Second degree burns – blister formation – very painful – damage to basement membrane – epidermis & superficial dermis 3. Third degree burns – dermis – painless – aka = full thickness burns 4. Fourth degree burns – subcutaneous tissue – muscle & bone A mixture is of the above is a common finding
  • 5. DEPENDING ON THICKNESS OF SKIN INVOLVED I. Partial thickness burns: It is either first or second degree burn which is red and painful, often with Blisters II. Full thickness burns: It is third degree burns which is charred, insensitive, deep involving all layers of the skin.
  • 7. PATHOPHYSIOLOGY Heat causes coagulation necrosis of skin and subcutaneous tissue. Release of vasoactive peptides Altered capillary permeability Loss of fluid Severe hypovolemia Decreased cardiac output 1. Decreased renal blood -Oliguria (Renal failure) 2. Altered pulmonary resistance causing pulmonary oedema  Infection  Systemic Inflammatory Response Syndrome (SIRS)  Multi Organ Dysfunction Syndrome (MODS).
  • 8. PHASE 1 = RAPID RESPONSE SYSTEM = NERVOUS SYSTEM • “Fight or flee” • Governed by hypothalamus = autonomic nervous system [sympathetic component] • Release of adrenaline and noradrenaline • Pupils dilate • Heart rate rises • Brain alert • Airways dilate
  • 9. PHASE 2 = LOCAL RESPONSE • Initial response is vasoconstriction by the catecholamine augmented by endothelin = most potent vasoconstrictor known • Soon this is replaced by vasodilatation and increased vascular permeability at injury site • Principal vasodilators include nitric oxide, prostacyclin, histamine, serotonin • This allows for the extravasation of inflammatory cells to come in to kill the invaders and clean up the mess
  • 10. PHASE 3 = ENDOCRINE RESPONSE • Remember the bigger the insult or injury the bigger the response • Therefore limited injury will have no systemic response • The goal of all these responses is preserve intravascular volume • Any insult threatening intravascular integrity HYPOTHALAMUS-PITUITARY-ADRENAL AXIS • Low Bp is picked up by the baroreceptors => hypothalamus=>corticotrophin releasing hormone=> adrenacorticotropin hormone=> cortisol & aldosterone=> raises sensitivity of catecholamine & sodium reabsorption
  • 11. RENIN-ANGIOTENSIN-ALDOSTERONE AXIS • Baroreceptors in the juxtaglomerular apparatus=> renin=> angiotensinogen=> angiotensin-1=> angiotensin converting enzyme=>angiotensin-2=> vasoconstrictor & stimulate aldosterone release by the adrenal cortex
  • 12. CRITERIA FOR ADMISSION TO A BURNS CENTER 1. Burns >20% bsa in adults 2. Burns >10% bsa in children & elderly >50 3. Electrical burns 4. Chemical burns [extensive] 5. Suspected child abuse burns 6. Inhalation burns 7. Special areas – face, hands, genitalia , major joints 8. Concomitant trauma – trauma center first 9. Co-morbidities – cardiopulmonary, dm, epilepsy 10. >5% deep burns
  • 13. ASSESMENT OF BURNS 1. WALLACE RULE OF 9
  • 14. RULE OF PALM • “RULE OF THE PALM” = PATIENT’S PALM IS APPROXIMATELY 1% OF THE BSA IN BOTH ADULTS AND CHILDREN
  • 15. INVESTIGATIONS • FBC. • U+E. • If inhalation suspected: chest X-ray, arterial blood gases, CO • estimation. • Blood group and crossmatch. • ECG/cardiac enzymes with electrical burns.
  • 16. IN SUMMARY 1. ABCDE 2. Pain control 3. Tetanus prophylaxis 4. Secondary survey + % bsa + depth 5. Foley catheter 6. Fluid resuscitation => oral or intravenous 7. Proton pump inhibitors 8. Topical antibiotics 9. Daily wound care 10. Keep the room warm
  • 17. OTHER ESGENERAL MEASUR • High protein diet • Blood transfusion • Physiotherapy • The use of a cradle • Always keep the patient warm
  • 18. FLUID RESUSCITATION 1. PARKLAND FORMULA:- 1. 4ml x KG x % BURN AREA = MLS 2. Half is administered in first 8 hours 3. The next half is administered in the following 16 hours • This formula or any formula is not cast in stone and should be modified to achieve the desired urine output • Literature says don’t go beyond 8.4 liters. Maximum percentage for fluid should be less than 50% • But in practice (30-40%) E.g. 22years old student at Zera sustains 20% burns, he weight is 70 calculate his fluid requirement using parkland formula
  • 19. 2. MUIR AND BARCLAY • =(%BSA x Kg) /2 Fluids are given over 36 hrs. as following
  • 20. 3. MODIFIED BROOKES It calculates the deficity: =2 X %bsa x kg.
  • 21. COMPLICATIONS OF BURNS Early complications • Airway obstruction • Shock • Hypoxia • RDS • Hypothermia Intermediately complications • Electrolyte imbalance • Infection • Anemia • Stress ulcers (Curling ulcers) • Paralytic ileus • Renal failure • Compartment syndrome
  • 22. LATE COMPLICATIONS • Contractures • Hypertrophic scar • keloids • Margolis's ulcer