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JAGDISH. K PROF. DR. A. GOWRISHANKAR’S UNIT ECG OF THE WEEK
30 yr old male was brought with the chief complaints of fever, altered sensorium for the past 3 days. Patient was apparently alright 3 days ago. Known type 1 diabetic on Insulin.
Examination  Patient was febrile, dyspneic, tachypneic, disoriented Pulse : 110/min BP : 100/60 mm of hg RR : 38/min CNS :  disorientation + Neck rigidity + Other systems : normal
ECG
ECG changes in electrolyte imbalance is not very predictable. Change depends on the inter individual variation & on the other electrolytes too.
However certain ECG features often develop in conjunction with increased / decreased potassium concentration so that ECG may be frequently utilised for electrolyte disturbances  particularly if the interpreter is thinking of electrolyte disturbances.
Accuracy of observation is enhanced if  Control tracing available Serial tracings done
Hyperkalemia  It has not been definitely established that the changes are due to Intracellular K +  changes Gradient across cell membrane Potassium level in the serum alone
Action Potential Atrial
SA Nodal
Ventricular
Similarities vs Differences
K +  changes & Action Potential curve
Changes during depolarisation & resting membrane potential
Changes during repolarisation
ECG changes Short QT interval Peaked T wave ST segment depression Lowering & widening of P wave Prolonged PR interval
QRS widening Short R wave & deep S wave Marching of QRS & T wave towards each other The Nadir of S wave & peak of T wave is connected by a straight line. Sine wave pattern Atrial fibrillation & Atrial arrest
Sinoventricular rhythm
Effect of other electrolytes Low Na + High Na +
Low Ca 2+ High Ca 2+
ECG
Sine wave
 

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ECG: Hyperkalemia