Evaluation of Low Back Pain (Ray).ppt
- 1. Evaluation of Low Back Pain
- 2. Introduction
- 5. Most disc herniations occur at L5-S1 At least 30% of the healthy symptomless population have clinically significant disc protrusions (Stadnik et al., 1998). What is Back Pain ?
- 6. What is Back Pain ? Several studies have shown that there is no correlation between MRI findings and patients’ low back symptoms. 1. Wittenberg et al., 1998 2. Smith et al., 1998 3. Savage et al., 1997
- 7. What is Back Pain ? There are many more joints in the back than discs. There are many more muscles than joints. The most common cause of low back pain is when one or more muscles “forget” to relax. We call this a somatic dysfunction.
- 8. Common Sources of LBP Somatic dysfunction Muscle in “spasm” Nerve root In somatic dysfunction, some muscles become overactive (“spasm”) and other muscles become inactive.
- 9. Common Sources of LBP Any dysfunction involving the thoracic or lumbar spine, the sacroiliac joint or the hip can create low back pain.
- 10. Common Sources of LBP L2 L3 L4 L5 S1 S2
- 11. Long dorsal si ligament sacrotuberous ligament sacrospinous ligament sciatic nerve piriformis Common Sources of LBP
- 12. Role of the sacroiliac joint The coxal bones consist of a thin shell of cortical bone (1-2 mm) over trabecular bone. Muscles play an important role in helping the pelvis resist stress. When muscles can’t work due to pain, the risk of injury increases.
- 13. Back Facts
- 14. Introduction • COMMON, 2ND only to URTI • Tx is symptomatic • HISTORY is critical to ruling out serious issues. • Conduct a Physical Exam to confirm and assess functional status
- 15. • What Causes Acute Low Back Pain – Muscle strain? – DJD or OA? – Disc disease? – Who cares? • Initially they are all treated same for the most part. • Most all get better with conservative treatment. • Beware of the serious causes!
- 16. Evaluate for “Red Flags”: May Signal Serious Causes of LBP • Cancer • Infection • Fracture • Sciatica • Cauda Equina syndrome • Ankylosing spondylitis
- 17. Sciatica • The sciatic nerve is the longest nerve in your body. It runs from your spinal cord to your buttock and hip area and down the back of each leg. The term "sciatica" refers to pain that radiates along the path of this nerve — from your back down your buttock and leg. Source: Mayoclinic.com
- 18. Cauda Equina Syndrome: • Caused by massive midline disc herniation or mass compressing cord or cauda equina. – Rare (<.04% of LBP patients). – Needs emergent surgical referral. • Symptoms: bilateral lower extremity weakness, numbness, or progressive neurological deficit. • Ask about: – Recent urinary retention (most common) or incontinence? – Fecal incontinence?
- 19. Ankylosing spondylitis • Ankylosing spondylitis is one of many forms of inflammatory arthritis, the most common of which is rheumatoid arthritis. Ankylosing spondylitis primarily causes inflammation of the joints between the vertebrae of your spine and the joints between your spine and pelvis (sacroiliac joints). Source: Mayoclinic.com
- 20. Evaluation of the Patient With LBP • Start with a detailed history – your best diagnostic tool. – Get an idea of the severity. – Look for the “red flags” of serious causes. • Use the physical exam to confirm what you suspect based on history. • Keep in mind: – Most of the time you won’t have a definitive diagnosis. – Imaging rarely changes initial treatment. – Most patients get better with conservative TX.
- 22. What Was the Mechanism of Injury or Overuse? • Was there an acute trauma or injury? – Sudden severe pain with bending. – Motor vehicle accident or fall. • Was there a recent history of excessive lifting or bending?
- 23. • About 85-90% of LBP sufferers will get better in 3 days to 6 weeks –Most back problems are not surgical cases • Of the remaining 10-15%, most will never get completely well
- 25. Mechanisms of Injury • Congenital abnormalities • Poor body mechanics • Back trauma
- 26. Pathology of Low Back Pain • Causes: –Herniated disks, facet pathology, spinal stenosis, stress fractures (spondys), compression fractures, ligamentous sprains, adaptive shortening, and muscle strain • Do spinal abnormalities always cause low back pain? –MRIs on 98 people with no back pain • Dr. Maureen Jensen, Hoag Memorial Hospital, Newport Beach, CA. (1995) –Nearly 2/3 had spinal abnormalities including bulging or protruding discs
- 28. Nerves • Spinal Nerves and Plexi – 31 spinal nerves – 4 Plexi • Cervical • Brachial • Lumbar (T12-L5) – Femoral, Obturator • Sacral (L4-S5) – Sciatic » Tibial and Common Peroneal
- 30. Neural Testing Dermatomes -correspond to an area of skin that is innervated by the cutaneous neurons of a single spinal nerve or cranial nerve. Myotomes -correspond to groups of muscles innervated by a specific nerve root.
- 32. Classification
- 33. Classify patient • Determine cause of problem – Postural • Inflammation of soft tissues – Dysfunctional • Adaptive Shortening • Strain or Sprain – Derangement • Disk • Facet joint • Stress Fracture
- 34. Sprain/Strain Dysfunction/ Postural Derangement ONSET Sudden, simple move Gradual Sudden, simple move PAIN Severe ache, diffuse, spasm Ache, intermittent Sharp, burning, Localized or Radiating MOBILITY Reduced, movement increases pain Reduced and stiffness Guarded flexion, extension decreases pain GOALS OF TX Decrease pain Decrease spasm Restore ROM Decrease pain Increase ROM Posture Strength/Flex Decrease pain Centralize disc Prevention Guide to Lumbar Spine Conditions
- 35. Lumbar Spine Conditions • Low Back Muscle Strain – Acute (Overextension) and Chronic (Faulty posture) • Facet Joint Dysfunction – Dislocation or Subluxation (Acute or Chronic) • Low Back fracture – Compression, Stress, or Spinous and Transverse Processes • Herniated Disc – Protrusion, Prolapse, Extrusion, and Sequestration – Local and Radiating Pain • Classic term “Sciatica”
- 36. Lumbar Spine Conditions • Spondylolysis – Unilateral defect in the pars interarticularis • Spondylolisthesis – Bilateral defect in the pars interarticularis which causes forward displacement of vertebra. • Spina Bifida Occulta – Congenital condition – spinal cord is exposed = delays in development.
- 37. Sacroiliac Joint Conditions • Sacral torsion – Forward or Backward torsion • Ilium torsion, upslip, downslip, outflare, inflare • Piriformis strain/trigger points
- 38. Unique risk factors for athletes • High impact trauma: – football, rugby • End range loading: – gymnastics, diving • Overuse trauma: – impact loading: distance running – rotational loading: golf, baseball – prolonged sitting: travel
- 39. Evaluation Techniques • HOPS/HIPS – History, Observation/Inspection, Palpation, Special Tests • Your first priority! – Establish the integrity of the spinal cord and nerve roots – History and several specific tests provide information (Dermatomes, Myotomes, Reflexes)
- 40. Assessing the Low Back – Primary Survey • Level of consciousness/Movement • Neurological system intact? – Secondary Survey • Pain, Dermatomes, Myotomes • ROM – only if no motor or sensory decrements • Further assessment on sidelines
- 41. Assessing the Low Back – HISTORY!!!! – Observation and Palpation • The Triad of Assessment – Asymmetry, ROM alteration, Tissue texture – Special Tests • Begin to be selective in you choices. • Classify tests as to their main findings • Use results of key tests to determine further testing
- 42. Triad of Assessment • Asymmetry – ASIS, PSIS, iliac crests, malleoli, feet • Range of motion alterations – Standing and seated flexion tests – Single leg stance test (Stork) – Springing of facet and sacroiliac joints – Guarding of certain positions • Tissue texture abnormalities – Muscles – “tootsie roll”
- 43. Specific evaluation techniques 1. HISTORY!!!! 2. Alignment and symmetry 3. Lumbar spine active movements 4. Neurological Testing 5. Disc Pathology Tests 6. Extension mechanics – Prone assessment 7. Sacroiliac tests 8. Sitting forward flexion and hip flexion 9. Standing forward flexion and hip flexion 10. Flexibility testing 11. Feet alignment
- 44. History • Location of pain • Onset of pain – Acute, chronic, or insidious • Mechanism of Injury (MOI) • Consistency of the pain – Constant vs. Intermittent pain • Bowel and Bladder signs • Changes in activity, surface, or equipment
- 45. What positions bother you? • Bending • Sitting • Rising from sitting • Standing • Walking • Lying prone • Lying supine
- 46. Evaluation Techniques • Observation/Inspection – Posture! – Range of motion • AROM • PROM • Observe their mechanics as they enter the room, get on table, remove shirts or shoes
- 47. Evaluation Techniques • Palpation – This is your chance to “contain” the injury to specific structures. – Also allows for natural comparison of “normal” landmarks • Muscular Tension – “Tootsie Roll Test” • Ligamentous Tests – Spring Test
- 48. Special Tests • Are they malingering? – Hoover’s Test • Determine whether injury is associated with intervertebral disc, nerve root, dural sheath, or bony deformity. • Positive tests for disc, nerve, or bony deformity ALWAYS warrant a referral to a physician
- 49. Tests for Nerve Root Impingement • Valsalva test • Milgram test • Kernigs/Brudzinski’s test • Straight Leg Raise – Affected and Well • Quadrant test • Slump test
- 50. Lumbar Spine Conditions • Low Back Muscle Strain – Very common and self-limiting – Acute (Overextension) and Chronic (Faulty posture) – Pain increases with passive and active flexion and resisted extension – Key Evaluative techniques: • History and Palpation • Rule out neural involvement • Test PROM, AROM, and RROM
- 51. Lumbar Spine Conditions • Low Back fracture – Compression or Stress – Body, Spinous Process, and Transverse Processes – Localized or diffuse pain – Treatment doesn’t relieve symptoms – X-ray and MRI are definitive diagnoses
- 52. Lumbar Spine Conditions • Facet Joint Dysfunction – Inflammation, sprain, degeneration – Dislocation or Subluxation (Acute or Chronic) • “stuck open” or “stuck closed” • Usually localized but may involve several segments • May be associated with nerve root impingement • Often times pain decreases with activity
- 53. Facet Joint Dysfunction • AROM – Flexion = “opening” and Extension = “closing” – Lumbar facet joints “open” on right side with left lateral flexion and left rotation – Lumbar facet joints “close” on right side with right lateral flexion and right rotation • Prone assessment – elbows to hands • Spring test • Quadrant test
- 55. Lumbar Spine Conditions • Herniated Discs – MOI: Overload (Direct or Indirect) or faulty biomechanics (or both) – Protrusion, Prolapse, Extrusion, and Sequestration – Pain usually aggravated by activity – Prolonged body position often increases symptoms • Patient may choose a position that relieves pain – Local and Radiating Pain • Reflexes and Sensory/Motor screening is essential – Definitive diagnosis comes from MRI
- 56. Disc and nerve root relationship
- 58. Neural Testing • Dermatomes • Myotomes – L1/L2 – Hip flexion – L3/L4 – Knee extension – L4 – Ankle dorsiflexion – L5 – Great toe extension – S1 – Eversion – S2 – Knee flexion
- 59. Observation • Posture – Plum line • Motions – Flexion – Extension – Lateral flexion – Rotation
- 62. Discogenic Pain • Special Tests: – Lower and Upper quarter screening • Dermatomes and Myotomes – Valsalva test – Milgram test – Well straight leg raise – Kernig’s/Brudzinski test – Quadrant test
- 64. Lumbar Spine Conditions • Sciatica – General term for inflammation of sciatic nerve – Sciatica is a result and NOT an injury in and of itself • Need to find what has caused the irritation – Disc, Muscle, Spondylopathy – Special tests: • Straight leg raise • Tension sign (Bowstrings) • Slump Test
- 65. Lumbar Spine Conditions • Spondylopathies – Mechanisms – Hyperextension • Onset – Insidious • Muscular imbalances – Pain usually localized (may radiate) • Increased during and after activity – Single leg stork stand • Unilateral – Pain with opposite leg – MRI or X-ray are definitive diagnoses
- 66. Spondylosis • Spondylolysis – generally mean changes in the vertebral joint characterized by increasing degeneration of the intervertebral disc with subsequent changes in the bones and soft tissues. – Unilateral or bilateral stable defect in the pars interarticularis – “Collared Scottie dog” deformity
- 67. Spondylolisthesis • Bilateral defect in the pars interarticularis which causes forward displacement of vertebra. • “Decapitated Scottie dog” deformity • “Step off deformity” • Adolescents and women
- 68. Spondys • Treatment: – REST and ice – Flexion is best. – Reduce extension moments. – Bracing sometimes a solution.
- 69. Sacroiliac Conditions • Hip, Ilium, and Sacral problems can stand alone OR • Can be connected to low back symptoms. – Cause or effect?
- 70. CAUSE or EFFECT? • Pelvis or Sacral alignment • Hamstring Tightness – Straight Leg Raise – 90/90 test • Hip Flexor tightness – Thomas Test – Trigger points • Piriformis tightness – IR of hip is limited – Trigger points
- 71. Special Tests for Pelvis and Sacrum • Alignment – Supine and prone – Prone extension • Sitting forward flexion and hip flexion – Monitoring PSIS – Monitoring low back • Standing forward flexion and hip flexion – Monitoring PSIS – Monitoring low back – Long Sitting Test – Pen Dot Test – FABERE – Gaenslen’s – Compression/Distraction – Outflare/Inflare
- 72. Pelvis and Sacral Conditions PELVIS • Upslip – ASIS and PSIS higher • Anterior Rotation – ASIS lower, PSIS higher • Tight hip flexor, weak gluteus • Posterior Rotation – ASIS higher, PSIS lower • Tight piriformis/gluteus and weak hip flexor SACRUM • Flexion – sulcus is deep • Extension – sulcus is shallow • Forward Torsion • Backward Torsion
- 73. ANY QUESTIONS ??
- 74. THANK YOU