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EXODEVIATIONS
Dr Abida Fasahtay
EXODEVIATION
• EXO-
Visual axis is deviated laterally
Fovea rotated nasally
EXODEVIATION = DIVERGENT STRABISMUS
Latent
(controlled by fusion)
Manifest:
-Intermittent or constant
-Unilateral or alternating
PSEUDOEXOTROPIA
• Appearance of exodeviation
Wide interpupillary distance
Large positive angle kappa- hypermetropia, retinopathy of
prematurity, toxocara canis retinitis.
TYPES
A. Concomitant
1. Primary
• Infantile exotropia
• Intermittent exotropia
2. Secondary
• Sensory exotropia
• Consecutive exotropia
B. Incomitant
1. Paralytic
2. Restrictive
3. Musculofascial innervational anomalies
ETIOLOGY
• Exact etiology unknown
• Proposed causes:
Excessive tonic divergence
Anatomical and mechanical factors within the orbit
MAGICIAN’S FORCEPS
PHENOMENON
• Abnormal proprioceptive impulses
originating from the dominant eye
as a cause of exodeviation as
concluded by Mitsui
• But unlikely that a mechanism
other than a visually elicited fixation
reflex accounts for the
phenomenon
1. DUANE’S CLASSIFICATION
• Merely a descriptive classification
1. Divergence excess pattern
2. Basic exodeviation
3. Convergence insufficiency pattern
4. Simulated divergence excess pattern
Difference of atleast 15 prism diopters
2. CLASSIFICATION ON THE BASIS OF
UNDERLYING FUSIONAL RESERVE
1.Exophoria : XP
2.Intermittent exotropia : X(T)
3.Manifest exotropia : XT
PRIMARY EXODEVIATIONS
CLINICAL CHARACTERISTICS
• Latent or intermittent form ↑
• Prevalence less than esodeviations
• Age of onset of the majority is shortly after birth
• Genuine “congenital” exotropia : poor prognosis
PROGRESSION
1. Divergence excess pattern → Stable
2. Basic exodeviation → Increased deviation or secondary
convergence insufficiency
3. Convergence insufficiency pattern→ binocular function
degenerates rapidly
4. Simulated divergence excess pattern → Increased near
deviation
SIGNS AND SYMPTOMS
• EXOPHORIA:
Eyestrain
Headache
Blurring of vision
Difficulties with prolonged periods of reading
Diplopia
• CHILDREN WITH INTERMITTENT OR CONSTANT
EXOTROPIA:
Less frequently symptomatic
• ADULTS WITH INTERMITTENT EXOTROPIA:
Commonly symptomatic
PHOTOPHOBIA
• Common in intermittent exodeviations
Child is outdoors & looking at infinity
No stimulus for convergence
Bright sunlight dazzles the retinas
Disruption of fusion
Deviation becomes manifest
Diplopia and confusion
Child closes one eye
MICROPSIA
• Less well known symptom
• Occurs when patient uses accomodative convergence to
control exodeviation at distance
SPECIAL TESTS : POST OCCLUSION
TEST OF SCOBEE-BURAIN
DIVERGENCE
EXCESS
NEAR
DEVIATION
AFTER
UNILATERAL
OCCLUSION
TRUE LITTLE
INFULENCE
SIMULATED INCREASE
SPECIAL TESTS : +3.00D SPHERICAL
LENS TEST
• +3.00D spherical lenses suspend accommodation and
thus accommodative convergence
EXOTROPIA NEAR DEVIATION USING
+3.00D LENS
LOW AC/A RATIO LITTLE INFLUENCE
HIGH AC/A RATIO INCREASE
MEASUREMENTS OF THE DEVIATION
• Unless the target used for distance fixation forces patients
to relax accommodation, and with it convergence, true
deviation of the eyes at distance fixation may remain
concealed.
• Therefore, measure angle while patient reads the 6/9 line
on the visual acuity chart
SENSORYADAPTATIONS
• Rare
• Strabismic amblyopia : almost non-existent
• Abnormal retinal correspondence : rarely seen
• Alternate suppression : Alternate exotropia
THERAPY
• Exophoria without asthenopia : No Rx
• Symptomatic exophoria and intermittent and constant
exodeviations : Usually Surgery
However, nonsurgical measures may be indicated to
create optimal sensory conditions before surgery
NONSURGICAL TREATMENT
SPECTACLE CORRECTION :
• Astigmatism and anisometropia : corrected
• Myopia : fully corrected
• Hypermetropia : correction depends upon its degree and
patient age
As a rule, a hypermetropia of less than +2.00DS in
children : do not correct
• Presbyopia : correct any underlying hypermetropia ,
weakest bifocal lens, base in prisms for near vision
NONSURGICAL TREATMENT
USE OF MINUS LENSES :
• High AC/A ratio : minus lenses
• Convergence insufficiency pattern : minus lenses
prescribed as lower segment bifocals
• Divergence excess pattern : minus lenses prescribed as
upper segment bifocals
PRISMS :
• Base in prisms
NONSURGICAL TREATMENT
• ORTHOPTICS :
• Supplement to surgery
• Convergence exercises and occlusion
SURGICAL TREATMENT
• Manifest exotropia present at or shortly after birth with no
history of intermittency
↓
Surgery performed as soon as reliable and constant
measures can be obtained
• Large angle constant exodeviation in adults
↓
Surgery performed as soon as diagnosis made
• Exophoria with asthenopia
↓
Surgery only of deviation not controlled by prisms
• Intermittent exotropia or constant exotropia preceded by a long period
of intermittency
Observation
-Occasional manifest squint
-Asymptomatic
-No progression
No Surgery
-Manifest squint ˃ 50% of waking hours
-Asthenopic symptoms
-Progression
Surgery
The most desirable age at which surgery should be performed for
intermittent exodeviations has been a matter of some dispute
Majority advocate delaying surgery until the child has
reached at least 4 years of age.
Rapid functional deterioration of
fusional control inspite of
nonsurgical Rx
Surgery at an
earlier age should
be considered
Finally, the size of deviation determines the decision to operate
FUNCTIONAL POINT OF VIEW Atleast 15 prism diopter
COSMETIC POINT OF VIEW Atleast 20-25 prism diopter
GOALS OF SURGERY
• For intermittent exotropia, small surgical overcorrection
(10-20 prism diopter) is desirable :
1. Divergent strabismic eyes show a strong tendency to
revert to their former position
2. Postoperative diplopia → fusional vergences → Stabilize
eventual alignment of eyes
• Lesser degrees of overcorrection → recurrence of
exodeviation
• Higher degrees of overcorrection → necessitate further
surgery for consecutive esotropia
CHOICE OF PROCEDURE
TRUE DIVERGENCE EXCESS B/L lateral rectus recession
SIMULATED DIVERGENCE EXCESS
OR BASIC EXOTROPIA
U/L recession-resection of lateral and
medial recti of nondominant eye
CONVERGENCE INSUFFICIENCY B/L medial rectus resection
Recession of lateral rectus muscle is more effective in
reducing the deviation at distance fixation than the
deviation at near
SURGICALRECESSION OF LATERAL RECTUS MUSCLE
SURGICAL RESECTION OF MEDIAL RECTUS MUSCLE
3-6 mm depending upon the size of deviation
MANAGEMENTOF OVERCORRECTIONS:
(CONSECUTIVE ESOTROPIA)
1. Large ovecorrection with gross limitation of ocular
motility
Immediate Surgery
2. Small overcorrection
Comitant Incomitant
Miotics
Plus lenses
Prism
Surgery
Surgery
MANAGEMENT OF UNDERCORRECTIONS:
Usually surgery
SECONDARY EXODEVIATIONS
SENSORY EXOTROPIA
• Primary sensory deficit in one eye:
Anisometropia
Cornea opacities
Dense cataract
Aphakia
Optic atrophy
Central macular scars etc
Disruption of fusion
• Correction of the visual deficit if possible
• Surgical correction : cosmetic
SECONDARY EXODEVIATIONS
CONSECUTIVE EXOTROPIA
• Formerly esotropic patient
• Either spontaneously or after surgical overcorrection
• Treatment : Surgery ( cosmetic )
INCOMITANT STRABISMUS
PARALYTIC
• 3rd nerve palsy
• Internuclear ophthalmoplegia (INO)
• Ocular myasthenia
Right INORight 3rd nerve palsy Ocular Myasthenia
INCOMITANT STRABISMUS
RESTRICTIVE
• Dysthyroid orbitomyopathy
• Fibrosis secondary to orbital trauma and orbital surgery
• Parasitic cyst
• Orbital tumors
 Duanes’s retraction syndrome type 2 :
• Lateral rectus innervation present on abduction as well as
adduction
• Abduction:
- normal
• Adduction:
-limited
-globe retraction
-narrowing of palpebral aperture
-upshoot or downshoot
INCOMITANT STRABISMUS
MUSCULOFASCIAL INNERVATIONAL ANOMALIES
 Duanes’s retraction syndrome type 2 :
• Treatment:
- Results of surgery disappointing
- Indication : significant deviation in primary position or
intolerable anomalous head position
- Ipsilateral recession of lateral rectus muscle
CONVERGENCE INSUFFICIENCY
• Remote near point of convergence and poor near fusional
vergence amplitudes
• Older child or adult
• Asthenopic symptoms
• Exophoria at near but not exotropia
• D/D convergence insufficiency type of exotropia
• Treatment :
- Correction of refraction
- Orthoptic exercises
- Base out prisms
- If these fail, base in prism reading glasses
- Medial rectus resection : rare cases
CONVERGENCE PARALYSIS
• Little if any fusional vergence amplitude
• Usually secondary to intracranial lesion
• Exotropia and diplopia on attempted near fixation only
• Adduction and accomodation are normal
• Distinct from convergence insufficiency:
- Acute onset
- Inability to overcome any base out prism
• Treatment :
- Base in prism at near
- Occlusion of one eye at near
- Eye muscle surgery is contraindicated
THANK YOU
EXOTROPIA : amount of surgical
correction
Deviation in
prism diopter
Recess LR
Both eye
mm
Resect MR
Both eye
mm
Recess LR + Resect MR
One eye
mm
15 4.5 3.0 4.0 3.0
20 5.0 3.5 5.0 3.5
25 5.5 4.0 5.5 4.0
30 6.0 4.5 6.0 4.0
35 6.5 5.0 6.5. 4.5
40 7.0 5.5 7.0 5.0
50 7.5 6.0 7.5 5.5
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Exodeviations

  • 2. EXODEVIATION • EXO- Visual axis is deviated laterally Fovea rotated nasally EXODEVIATION = DIVERGENT STRABISMUS Latent (controlled by fusion) Manifest: -Intermittent or constant -Unilateral or alternating
  • 3. PSEUDOEXOTROPIA • Appearance of exodeviation Wide interpupillary distance Large positive angle kappa- hypermetropia, retinopathy of prematurity, toxocara canis retinitis.
  • 4. TYPES A. Concomitant 1. Primary • Infantile exotropia • Intermittent exotropia 2. Secondary • Sensory exotropia • Consecutive exotropia B. Incomitant 1. Paralytic 2. Restrictive 3. Musculofascial innervational anomalies
  • 5. ETIOLOGY • Exact etiology unknown • Proposed causes: Excessive tonic divergence Anatomical and mechanical factors within the orbit
  • 6. MAGICIAN’S FORCEPS PHENOMENON • Abnormal proprioceptive impulses originating from the dominant eye as a cause of exodeviation as concluded by Mitsui • But unlikely that a mechanism other than a visually elicited fixation reflex accounts for the phenomenon
  • 7. 1. DUANE’S CLASSIFICATION • Merely a descriptive classification 1. Divergence excess pattern 2. Basic exodeviation 3. Convergence insufficiency pattern 4. Simulated divergence excess pattern Difference of atleast 15 prism diopters
  • 8. 2. CLASSIFICATION ON THE BASIS OF UNDERLYING FUSIONAL RESERVE 1.Exophoria : XP 2.Intermittent exotropia : X(T) 3.Manifest exotropia : XT
  • 9. PRIMARY EXODEVIATIONS CLINICAL CHARACTERISTICS • Latent or intermittent form ↑ • Prevalence less than esodeviations • Age of onset of the majority is shortly after birth • Genuine “congenital” exotropia : poor prognosis
  • 10. PROGRESSION 1. Divergence excess pattern → Stable 2. Basic exodeviation → Increased deviation or secondary convergence insufficiency 3. Convergence insufficiency pattern→ binocular function degenerates rapidly 4. Simulated divergence excess pattern → Increased near deviation
  • 11. SIGNS AND SYMPTOMS • EXOPHORIA: Eyestrain Headache Blurring of vision Difficulties with prolonged periods of reading Diplopia • CHILDREN WITH INTERMITTENT OR CONSTANT EXOTROPIA: Less frequently symptomatic • ADULTS WITH INTERMITTENT EXOTROPIA: Commonly symptomatic
  • 12. PHOTOPHOBIA • Common in intermittent exodeviations Child is outdoors & looking at infinity No stimulus for convergence Bright sunlight dazzles the retinas Disruption of fusion Deviation becomes manifest Diplopia and confusion Child closes one eye
  • 13. MICROPSIA • Less well known symptom • Occurs when patient uses accomodative convergence to control exodeviation at distance
  • 14. SPECIAL TESTS : POST OCCLUSION TEST OF SCOBEE-BURAIN DIVERGENCE EXCESS NEAR DEVIATION AFTER UNILATERAL OCCLUSION TRUE LITTLE INFULENCE SIMULATED INCREASE
  • 15. SPECIAL TESTS : +3.00D SPHERICAL LENS TEST • +3.00D spherical lenses suspend accommodation and thus accommodative convergence EXOTROPIA NEAR DEVIATION USING +3.00D LENS LOW AC/A RATIO LITTLE INFLUENCE HIGH AC/A RATIO INCREASE
  • 16. MEASUREMENTS OF THE DEVIATION • Unless the target used for distance fixation forces patients to relax accommodation, and with it convergence, true deviation of the eyes at distance fixation may remain concealed. • Therefore, measure angle while patient reads the 6/9 line on the visual acuity chart
  • 17. SENSORYADAPTATIONS • Rare • Strabismic amblyopia : almost non-existent • Abnormal retinal correspondence : rarely seen • Alternate suppression : Alternate exotropia
  • 18. THERAPY • Exophoria without asthenopia : No Rx • Symptomatic exophoria and intermittent and constant exodeviations : Usually Surgery However, nonsurgical measures may be indicated to create optimal sensory conditions before surgery
  • 19. NONSURGICAL TREATMENT SPECTACLE CORRECTION : • Astigmatism and anisometropia : corrected • Myopia : fully corrected • Hypermetropia : correction depends upon its degree and patient age As a rule, a hypermetropia of less than +2.00DS in children : do not correct • Presbyopia : correct any underlying hypermetropia , weakest bifocal lens, base in prisms for near vision
  • 20. NONSURGICAL TREATMENT USE OF MINUS LENSES : • High AC/A ratio : minus lenses • Convergence insufficiency pattern : minus lenses prescribed as lower segment bifocals • Divergence excess pattern : minus lenses prescribed as upper segment bifocals PRISMS : • Base in prisms
  • 21. NONSURGICAL TREATMENT • ORTHOPTICS : • Supplement to surgery • Convergence exercises and occlusion
  • 22. SURGICAL TREATMENT • Manifest exotropia present at or shortly after birth with no history of intermittency ↓ Surgery performed as soon as reliable and constant measures can be obtained • Large angle constant exodeviation in adults ↓ Surgery performed as soon as diagnosis made • Exophoria with asthenopia ↓ Surgery only of deviation not controlled by prisms
  • 23. • Intermittent exotropia or constant exotropia preceded by a long period of intermittency Observation -Occasional manifest squint -Asymptomatic -No progression No Surgery -Manifest squint ˃ 50% of waking hours -Asthenopic symptoms -Progression Surgery
  • 24. The most desirable age at which surgery should be performed for intermittent exodeviations has been a matter of some dispute Majority advocate delaying surgery until the child has reached at least 4 years of age. Rapid functional deterioration of fusional control inspite of nonsurgical Rx Surgery at an earlier age should be considered
  • 25. Finally, the size of deviation determines the decision to operate FUNCTIONAL POINT OF VIEW Atleast 15 prism diopter COSMETIC POINT OF VIEW Atleast 20-25 prism diopter
  • 26. GOALS OF SURGERY • For intermittent exotropia, small surgical overcorrection (10-20 prism diopter) is desirable : 1. Divergent strabismic eyes show a strong tendency to revert to their former position 2. Postoperative diplopia → fusional vergences → Stabilize eventual alignment of eyes • Lesser degrees of overcorrection → recurrence of exodeviation • Higher degrees of overcorrection → necessitate further surgery for consecutive esotropia
  • 27. CHOICE OF PROCEDURE TRUE DIVERGENCE EXCESS B/L lateral rectus recession SIMULATED DIVERGENCE EXCESS OR BASIC EXOTROPIA U/L recession-resection of lateral and medial recti of nondominant eye CONVERGENCE INSUFFICIENCY B/L medial rectus resection Recession of lateral rectus muscle is more effective in reducing the deviation at distance fixation than the deviation at near
  • 28. SURGICALRECESSION OF LATERAL RECTUS MUSCLE SURGICAL RESECTION OF MEDIAL RECTUS MUSCLE 3-6 mm depending upon the size of deviation
  • 29. MANAGEMENTOF OVERCORRECTIONS: (CONSECUTIVE ESOTROPIA) 1. Large ovecorrection with gross limitation of ocular motility Immediate Surgery 2. Small overcorrection Comitant Incomitant Miotics Plus lenses Prism Surgery Surgery MANAGEMENT OF UNDERCORRECTIONS: Usually surgery
  • 30. SECONDARY EXODEVIATIONS SENSORY EXOTROPIA • Primary sensory deficit in one eye: Anisometropia Cornea opacities Dense cataract Aphakia Optic atrophy Central macular scars etc Disruption of fusion • Correction of the visual deficit if possible • Surgical correction : cosmetic
  • 31. SECONDARY EXODEVIATIONS CONSECUTIVE EXOTROPIA • Formerly esotropic patient • Either spontaneously or after surgical overcorrection • Treatment : Surgery ( cosmetic )
  • 32. INCOMITANT STRABISMUS PARALYTIC • 3rd nerve palsy • Internuclear ophthalmoplegia (INO) • Ocular myasthenia Right INORight 3rd nerve palsy Ocular Myasthenia
  • 33. INCOMITANT STRABISMUS RESTRICTIVE • Dysthyroid orbitomyopathy • Fibrosis secondary to orbital trauma and orbital surgery • Parasitic cyst • Orbital tumors
  • 34.  Duanes’s retraction syndrome type 2 : • Lateral rectus innervation present on abduction as well as adduction • Abduction: - normal • Adduction: -limited -globe retraction -narrowing of palpebral aperture -upshoot or downshoot INCOMITANT STRABISMUS MUSCULOFASCIAL INNERVATIONAL ANOMALIES
  • 35.  Duanes’s retraction syndrome type 2 : • Treatment: - Results of surgery disappointing - Indication : significant deviation in primary position or intolerable anomalous head position - Ipsilateral recession of lateral rectus muscle
  • 36. CONVERGENCE INSUFFICIENCY • Remote near point of convergence and poor near fusional vergence amplitudes • Older child or adult • Asthenopic symptoms • Exophoria at near but not exotropia • D/D convergence insufficiency type of exotropia • Treatment : - Correction of refraction - Orthoptic exercises - Base out prisms - If these fail, base in prism reading glasses - Medial rectus resection : rare cases
  • 37. CONVERGENCE PARALYSIS • Little if any fusional vergence amplitude • Usually secondary to intracranial lesion • Exotropia and diplopia on attempted near fixation only • Adduction and accomodation are normal • Distinct from convergence insufficiency: - Acute onset - Inability to overcome any base out prism • Treatment : - Base in prism at near - Occlusion of one eye at near - Eye muscle surgery is contraindicated
  • 39. EXOTROPIA : amount of surgical correction Deviation in prism diopter Recess LR Both eye mm Resect MR Both eye mm Recess LR + Resect MR One eye mm 15 4.5 3.0 4.0 3.0 20 5.0 3.5 5.0 3.5 25 5.5 4.0 5.5 4.0 30 6.0 4.5 6.0 4.0 35 6.5 5.0 6.5. 4.5 40 7.0 5.5 7.0 5.0 50 7.5 6.0 7.5 5.5