Ethan J. Browning
  Derick D. Davis
What is stress?
   Stress is any condition that disturbs the
    physiological or psychological homeostasis
    of an organism

   Two types of stress:
     External stress is an external condition that puts
      a strain on the homeostatic mechanisms of the
      body
      ○ Example: excessive heat
     Internal stress is when perceived demands
      exceed perceived ability
      ○ Example: unrealistic expectations
Positive and Negative Stress
Positive Stress (Eustress)         Negative Stress (Distress)

   Stress is positive when it        Hinders performance
    is essential for survival or
    aids performance
                                      Chronic stress
   The primal function of
    stress is survival

                                      Can cause physical
   Motivation                         and/or mental damage
Seyle’s General Adaptation
Syndrome
    Three stage process that explains how
     individuals respond to stress

      First Stage – Alarm Reaction
       ○ Psychological
       ○ The body has become aware of the stress and
         prepares itself for the fight-or-flight response by
         releasing the hormones adrenaline and
         corticosteroid
       ○ Effects of the hormones include: increased heart
         rate, breathing rate, blood sugar level, and
         perspiration, and a decrease in digestion
       ○ Takes place in the Sympathetic System of the
         Autonomic Nervous System
Seyle’s General Adaptation
Syndrome
   Second Stage - Resistance Stage
    ○ The body attempts to cope with the stress
    ○ If the stress continues for a long period of
      time, the body will attempt to remain alert to
      be prepared to deal with the stressor
    ○ The body cannot continue in this stage for a
      longed period of time because its resources
      will gradually be depleted
    ○ Takes place in the Parasympathetic System of
      the Autonomic Nervous System
Seyle’s General Adaptation
Syndrome
   Third Stage - Exhaustion Stage
    ○ At this point, all of the body’s resources have
      been depleted and the body is no longer able
      to function normally
    ○ The symptoms that were caused in the alarm
      stage such as increased heart rate, breathing
      rate, blood sugar level, and perspiration, and
      a decrease in digestion may reappear
    ○ If the body experiences a prolonged
      exhaustion stage the immune system can be
      impaired and severe long term damage can
      result
Memory
   Memory is an organism's ability to store,
    retain, and recall information and
    experiences.

   There are many different models that
    explain what memory is and how it
    functions.
Information Processing
Perspective
   There are three main stages in the
    formation and retrieval of memory
     First Stage – Encoding
      ○ Information is received, processed, and
        combined with other received information
     Second Stage – Storage
      ○ The creation of a permanent record of the
        encoded information is formed
     Third Stage – Retrieval
      ○ Stored information is recalled using a cue
Types of Memory
   Three types of memory
     Sensory Memory is memorization or the ability to
      look at an object and remember what it looked like
      ○ Brief – lasts only 200 to 500 milliseconds after the item
        is observed
     Short-Term Memory can be recalled for several
      seconds to even a minute after the information has
      been observed
      ○ Capacity is limited
     Long-Term Memory information from sensory
      memory or short-term memory that has been
      transferred to long-term memory through repetition
      or other means
      ○ Unlimited in capacity and duration
Hippocampus and Memory
   The portion of the brain most responsible for
    memory is the hippocampus

   Located in the medial temporal lobe of the
    brain

   Responsible for converting information in the
    short-term memory into the long-term memory,
    and inhibition and spatial perception

   It is not yet known what exact role the
    hippocampus plays in memory
The Effects of Stress on
Memory
   Stress can affect various aspects of memory, brain
    structures, and physiological processes

   There have been many studies performed on both
    rodents and humans of how stress affects the
    hippocampus

   The hippocampus, which is a crucial component of
    memory, is highly susceptible to long-term stress
    than other portions of the brain
     Due to the hippocampus having a large quantity of
      corticosteroid receptors
     Stress hormones such as corticosteroid released over a
      long period of time damages the hippocampus
The Effects of Stress on
Memory
   The damage to the hippocampus can include:
    reduced excitability of hippocampal neurons,
    inhibited creation of new neurons, and atrophy of
    dendrites

   The result is that certain hippocampal functions such
    as learning and memory are impaired and damaged
    due to stress

   Some of these effects can be reversed if the stress is
    discontinued

   Some of the effects are irreversible
Studies
   Studies done over two decades support the correlation between stress,
    the hippocampus, and memory impairment

   Patients that suffer from Post-Traumatic Stress Disorder have
    hippocampal atrophy

   People diagnosed with depression and hypercortisolemia (a condition
    where an individual has high amounts of circulating cortisol) have
    memory impairments due to hippocampus damage

   Patients that suffer from Cushing’s disease (a disease in which tumors
    in the adrenal gland cause excess secretion of glucocorticoids) have
    hippocampal atrophy and memory impairment

   Studies conducted on rats that were exposed to stress or given
    corticosterone displayed impairments in spatial memory
Our study
   The objective of the study was to determine if there is a correlation
    between stress level and memory performance.

   Consisted of 11 participants, all over the age of 18, and current college
    students

   Participants were instructed to complete an Undergraduate Stress
    Questionnaire to access their stress level

   Participants were provided with a page that contained a series of
    pictures and were asked to study them for 30 seconds

    At the end of 30 seconds, the pages were collected, and the
    participants were instructed to write down as many of the pictures that
    they could recall onto the answer sheet

   The hypothesis was that short-term memory performance of
    undergraduates would decrease according to stress level.
Assessment
   Stress levels were accessed by the
    number of items the participant checked
    on the Undergraduate Stress
    Questionnaire

   Memory test scores were accessed by
    the number of correctly recalled pictures
Results
       The results do not support nor reject the
        hypothesis
                                            Memory Test
                        16

                        14
    Memory Test Score




                        12

                        10

                         8

                         6

                         4

                         2

                         0
                             0   10    20        30        40   50   60

                                      Stress Level Score
Discussion
   Although there is a slight downward trend in memory test
    scores of the participants that had higher stress level scores
    towards the end of the graph, the results are inconclusive.

   It is predicted that with a larger sample of participants that the
    hypothesis could be proven correct.

   The study had many other variables that could have interfered
    with the data.
     The Undergraduate Stress Questionnaire did not take into account other
      life stressors, therefore the participants stress levels could have been
      inaccurately assessed
     It is difficult to assess an individual’s stress level because the amount of
      stress that a situation creates varies with each individual’s
      perception, therefore the stress levels again could have been assessed
      inaccurately
     Participants were not screened for any learning or memory disabilities or
      difficulties
References
Glassman, A. (2009). How stress affects short term and long
   term memory. Journal of Professional Education, 12(5), 1-
   16.
Kim, J. J. & Diamond, D. M. (2002). The stressed
   hippocampus, synaptic plasticity and lost memories.
   Nature Reviews Neuroscience, 3, 453-462.
Merz, C.J. & Wolf, O. T. (2010). Stress impairs retrieval of
   socially relevant information. Behavioral
   Neuroscience, 124(2), 288-293.
Stegeren, A.H. (2009). Imaging stress effects on memory: a
   review of neuroimaging studies. The Canadian Journal of
   Psychiatry, 54(1), 16-27.
Woods, D. D. & Baker, A. M. (2005). Memory models for
   high-school psychology lesson plans. Psychology
   Teachers Journal, 27(6), 35-52.

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Group research assignment powerpoint

  • 1. Ethan J. Browning Derick D. Davis
  • 2. What is stress?  Stress is any condition that disturbs the physiological or psychological homeostasis of an organism  Two types of stress:  External stress is an external condition that puts a strain on the homeostatic mechanisms of the body ○ Example: excessive heat  Internal stress is when perceived demands exceed perceived ability ○ Example: unrealistic expectations
  • 3. Positive and Negative Stress Positive Stress (Eustress) Negative Stress (Distress)  Stress is positive when it  Hinders performance is essential for survival or aids performance  Chronic stress  The primal function of stress is survival  Can cause physical  Motivation and/or mental damage
  • 4. Seyle’s General Adaptation Syndrome  Three stage process that explains how individuals respond to stress  First Stage – Alarm Reaction ○ Psychological ○ The body has become aware of the stress and prepares itself for the fight-or-flight response by releasing the hormones adrenaline and corticosteroid ○ Effects of the hormones include: increased heart rate, breathing rate, blood sugar level, and perspiration, and a decrease in digestion ○ Takes place in the Sympathetic System of the Autonomic Nervous System
  • 5. Seyle’s General Adaptation Syndrome  Second Stage - Resistance Stage ○ The body attempts to cope with the stress ○ If the stress continues for a long period of time, the body will attempt to remain alert to be prepared to deal with the stressor ○ The body cannot continue in this stage for a longed period of time because its resources will gradually be depleted ○ Takes place in the Parasympathetic System of the Autonomic Nervous System
  • 6. Seyle’s General Adaptation Syndrome  Third Stage - Exhaustion Stage ○ At this point, all of the body’s resources have been depleted and the body is no longer able to function normally ○ The symptoms that were caused in the alarm stage such as increased heart rate, breathing rate, blood sugar level, and perspiration, and a decrease in digestion may reappear ○ If the body experiences a prolonged exhaustion stage the immune system can be impaired and severe long term damage can result
  • 7. Memory  Memory is an organism's ability to store, retain, and recall information and experiences.  There are many different models that explain what memory is and how it functions.
  • 8. Information Processing Perspective  There are three main stages in the formation and retrieval of memory  First Stage – Encoding ○ Information is received, processed, and combined with other received information  Second Stage – Storage ○ The creation of a permanent record of the encoded information is formed  Third Stage – Retrieval ○ Stored information is recalled using a cue
  • 9. Types of Memory  Three types of memory  Sensory Memory is memorization or the ability to look at an object and remember what it looked like ○ Brief – lasts only 200 to 500 milliseconds after the item is observed  Short-Term Memory can be recalled for several seconds to even a minute after the information has been observed ○ Capacity is limited  Long-Term Memory information from sensory memory or short-term memory that has been transferred to long-term memory through repetition or other means ○ Unlimited in capacity and duration
  • 10. Hippocampus and Memory  The portion of the brain most responsible for memory is the hippocampus  Located in the medial temporal lobe of the brain  Responsible for converting information in the short-term memory into the long-term memory, and inhibition and spatial perception  It is not yet known what exact role the hippocampus plays in memory
  • 11. The Effects of Stress on Memory  Stress can affect various aspects of memory, brain structures, and physiological processes  There have been many studies performed on both rodents and humans of how stress affects the hippocampus  The hippocampus, which is a crucial component of memory, is highly susceptible to long-term stress than other portions of the brain  Due to the hippocampus having a large quantity of corticosteroid receptors  Stress hormones such as corticosteroid released over a long period of time damages the hippocampus
  • 12. The Effects of Stress on Memory  The damage to the hippocampus can include: reduced excitability of hippocampal neurons, inhibited creation of new neurons, and atrophy of dendrites  The result is that certain hippocampal functions such as learning and memory are impaired and damaged due to stress  Some of these effects can be reversed if the stress is discontinued  Some of the effects are irreversible
  • 13. Studies  Studies done over two decades support the correlation between stress, the hippocampus, and memory impairment  Patients that suffer from Post-Traumatic Stress Disorder have hippocampal atrophy  People diagnosed with depression and hypercortisolemia (a condition where an individual has high amounts of circulating cortisol) have memory impairments due to hippocampus damage  Patients that suffer from Cushing’s disease (a disease in which tumors in the adrenal gland cause excess secretion of glucocorticoids) have hippocampal atrophy and memory impairment  Studies conducted on rats that were exposed to stress or given corticosterone displayed impairments in spatial memory
  • 14. Our study  The objective of the study was to determine if there is a correlation between stress level and memory performance.  Consisted of 11 participants, all over the age of 18, and current college students  Participants were instructed to complete an Undergraduate Stress Questionnaire to access their stress level  Participants were provided with a page that contained a series of pictures and were asked to study them for 30 seconds  At the end of 30 seconds, the pages were collected, and the participants were instructed to write down as many of the pictures that they could recall onto the answer sheet  The hypothesis was that short-term memory performance of undergraduates would decrease according to stress level.
  • 15. Assessment  Stress levels were accessed by the number of items the participant checked on the Undergraduate Stress Questionnaire  Memory test scores were accessed by the number of correctly recalled pictures
  • 16. Results  The results do not support nor reject the hypothesis Memory Test 16 14 Memory Test Score 12 10 8 6 4 2 0 0 10 20 30 40 50 60 Stress Level Score
  • 17. Discussion  Although there is a slight downward trend in memory test scores of the participants that had higher stress level scores towards the end of the graph, the results are inconclusive.  It is predicted that with a larger sample of participants that the hypothesis could be proven correct.  The study had many other variables that could have interfered with the data.  The Undergraduate Stress Questionnaire did not take into account other life stressors, therefore the participants stress levels could have been inaccurately assessed  It is difficult to assess an individual’s stress level because the amount of stress that a situation creates varies with each individual’s perception, therefore the stress levels again could have been assessed inaccurately  Participants were not screened for any learning or memory disabilities or difficulties
  • 18. References Glassman, A. (2009). How stress affects short term and long term memory. Journal of Professional Education, 12(5), 1- 16. Kim, J. J. & Diamond, D. M. (2002). The stressed hippocampus, synaptic plasticity and lost memories. Nature Reviews Neuroscience, 3, 453-462. Merz, C.J. & Wolf, O. T. (2010). Stress impairs retrieval of socially relevant information. Behavioral Neuroscience, 124(2), 288-293. Stegeren, A.H. (2009). Imaging stress effects on memory: a review of neuroimaging studies. The Canadian Journal of Psychiatry, 54(1), 16-27. Woods, D. D. & Baker, A. M. (2005). Memory models for high-school psychology lesson plans. Psychology Teachers Journal, 27(6), 35-52.